Taurin repariert all die kleinen Funktionseinbußen der Verschleimung – besonders was die zentralnervöse Steuerung angeht. Vergeßlichkeit, Antrieb und Konzentrationsfähigkeit bessern sich ebenso wie die Wachheit tagsüber.
Es ist ein Mittel gegen die Alterung als solche, was sich in einer Besserung von Symptomen an Haut und Schleimhäuten sowie der Nerven zeigt, also den schnell sich teilenden und empfindlichen Zellen, die von der Alterung als erste betroffen sind. Das Schwinden der Funktion der Sinnesorgane nimmt bei Taurinzufuhr ab, Entzündungsneigung mindert sich und Muskelabbau ebenso wie die Minderung der Hormonproduktion bessert sich. Vegetative Funktionen stabilisieren sich wie Blutdruck, Herzschlag, Schlaf, Kältetoleranz. Ablagerungen werden abgebaut und Degeneration als Folge der allgemeinen Funktionsminderung geht zurück.
Übersicht
- Metabolisches Syndrom
- KHK
- Arteriosklerose
- Herzinsuffizienz
- Adipositas
- Hyperhomocysteinämie
- Nervenschutz
- Muskelschwund
- Sexualhormonmangel
- Entzündungen
- Retinadegeneration
- Haarausfall
- Arthrosen
- Fibrosen
- Verletzungen
- Alterung
- Krebs
- Darmdysbiose
- Toxische Hepatitis
- Perioodontitis
- Nebenwirkungen Chemotherapie
- Osteoporose
- Pankreatitis
- Thrombosen
- Metabolisches Syndrom
The beneficial effects of taurine in preventing metabolic syndrome
Metabolic syndrome, a cluster of risk factors for diabetes and cardiovascular disease, has become a very serious public health concern. A number of studies have provided evidence that taurine has an efficient action against metabolic syndrome, which includes reducing triglycerides to prevent obesity, improving insulin resistance to regulate glucose metabolism, lowering cholesterol (especially decreasing VLDL + LDL cholesterol and increasing HDL cholesterol) to prevent diet-induced hypercholesterolemia, and regulating the renin-angiotensin-aldosterone system and the kallikrein-kinin system etc. to reduce blood pressure.
1.1. Diabetes
In addition, taurine and pioglitazone significantly decreased lipid peroxidation and DNA damage, and enhanced activity of the antioxidant enzymes in testes of diabetic rats. In conclusion, taurine and pioglitazone exerted protective effects against diabetes-induced testicular damage through attenuation of hyperglycemia, inflammation, oxidative stress and DNA damage, and up-regulation of the pituitary/gonadal axis.
Erectile function was significantly reduced in the diabetic rats compared with in the nondiabetic rats, and was improved in the diabetic rats treated with taurine. The corpus cavernosum of the rats with DED exhibited severe fibrosis and decreased smooth muscle content. Deposition of extracellular matrix proteins was increased in the diabetic rats, while expression of endothelial nitric oxide synthase/cyclic guanosine monophosphate/nitric oxide pathway-related proteins was reduced. Taurine supplementation ameliorated erectile response as well as histologic and molecular alterations.
Taurine also reversed the lower brain weight and improved the short-term memory in diabetic rats. Thus, the taurine antidepressant effect may be explained by interference with the GABA system, in line to its neuroprotective effect showed here by preventing brain weight loss and improving memory in diabetic rats.
Taurine also partially alleviated neuroinflammation as reflected by suppressed the NF-κB expression and enhanced the Nrf2, HO-1, GLUT1,3 expressions in the diabetic rats. In conclusion, taurine reduces the severity of oxidative stress through activating antioxidative defense signaling pathway in diabetic rat brain
1.2. Hypertonie
These findings suggest that oral supplementation of taurine may prevent the increase in SBP induced by DECA, an effect potentially mediated by angiotensin-converting enzyme.
In conclusion, the antihypertensive effect of chronic taurine supplementation shows promise in the treatment of prehypertension through improvement of vascular function.
1.3. Hypercholesterinämie
These observations suggest that taurine supplementation increases the synthesis and excretion of taurine-conjugated bile acids and stimulates the catabolism of cholesterol to bile acid by elevating the expression and activity of CYP7A1. This may reduce cholesterol esterification and lipoprotein assembly for very low density lipoprotein (VLDL) secretion, leading to reductions in the serum and hepatic cholesterol levels.
Taurine enhanced the serum HDL-cholesterol concentration in a dose-dependent manner without any change in total cholesterol
- KHK
Taurine and Mg supplementation increased EPC colony formation in healthy men and improved impaired EPC function in SHRs through antioxidation, indicating that the dietary intake of taurine and Mg may prolong lifespan by preventing the progression of cardiovascular diseases.
- Arteriosklerose
Diverse effects of taurine on vascular response and inflammation in GSH depletion model in rabbits
Treatment with BSO resulted in vascular reactivity changes and increased immunostaining of TNF-α in mainly carotid arteries in this model of oxidative stress. The effect of taurine on BSO-induced vascular reactivity changes varied depending on the vessel. The inhibition of the increase in TNF-α expression by taurine in both carotid arteries and aortae supports the proposal that taurine has a beneficial effect in the treatment of inflammatory diseases such as atherosclerosis.
- Herzinsuffizienz
After taurine was started, vital signs stabilized and lowering of dry weight was possible. In addition, X-ray and cardiac diastolic failure on echocardiography improved. Taurine was effective for CHF patients on HDF in whom dry weight was difficult to control in spite of various medications.
In the taurine-treated group significant treatment effect was observed on systolic left ventricular function after 6 weeks
Taurine has also attracted attention because it has beneficial actions in CHF, in part by its demonstrated inhibition of the harmful actions of the neurohumoral factors. In this review, we summarize the beneficial actions of taurine in CHF, focusing on its antagonism of the catecholamines and angiotensin II.
- Adipositas
MSG obesity in rats was not associated with alterations in pro-inflammatory markers in retroperitoneal fat stores; however, reductions in the serum concentrations of anti-inflammatory cytokines and of TNF-α were observed. TAU treatment decreased adiposity, and this effect was associated with the normalization of circulating TNF-α and IL-4 concentrations in MTAU rats.
Tau supplementation prevented obesity and hepatic TG deposition by upregulating MTP mRNA, ameliorating hepatic lipid efflux, and consequently enhancing PPAR-α which increases lipid oxidation through ACO and CPT-1a gene expressions.
Dietary intake of taurine and glycine correlated negatively with body mass gain and total fat mass, while intake of all other amino acids correlated positively. Furthermore taurine and glycine intake correlated positively with improved plasma lipid profile, i.e., lower levels of plasma lipids and higher HDL-to-total-cholesterol ratio. In conclusion, dietary scallop protein completely prevents high-fat, high-sucrose-induced obesity whilst maintaining lean body mass and improving the plasma lipid profile in male C57BL/6J mice.
- Hyperhomocysteinämie
Effect of taurine supplementation on plasma homocysteine levels of the middle-aged Korean women
The effect of taurine on the levels of plasma Hcy was assessed by regression analysis (R2 = 0.304). After taurine supplementation, plasma taurine and Hcy concentration exhibited a significant negative correlation (p < 0.05). In conclusion, taurine is an effective nutrient that antagonizes Hcy levels. Therefore, this study suggests that sufficient taurine intake might be an effective way of preventing cardiovascular diseases, such as atherosclerosis.
- Nervenschutz
We found that 100μM NMDA induced oxidative stress by increasing the reactive oxygen species level, which contributed to the cell death, in vitro. Neuron cultures pretreated with 25mM taurine showed lower percentage of death cells and declined reactive oxygen species level. Moreover, taurine attenuated Nox2/Nox4 protein expression and enzyme activity and declined intracellular calcium intensity during NMDA-induced neuron injury. Additionally, taurine also showed neuroprotection against H2O2-induced injury, accompanying with Nox inhibition. So, we suppose that protection of taurine against reactive oxygen species during NMDA-induced neuron injury is associated with Nox inhibition, probably in a calcium-dependent manner.
Both CAR and TAU diminished apoptosis and ameliorated histopathological findings in the brain of GAL-treated rats. Our results indicate that CAR and TAU may be effective to prevent the development of oxidative stress, apoptosis and histopathological deterioration in the brain of GAL-treated rats.
Our study supports that taurine may play important roles in the pathophysiology of PD and the disturbances caused by chronic levodopa administration.
Taurine had a direct effect on stem/progenitor cells proliferation, as observed in vitro, and also reduced activated microglia. Furthermore, taurine increased the survival of newborn neurons, resulting in a net increase in adult neurogenesis. Together, these results show that taurine increases several steps of adult neurogenesis and support a beneficial role of taurine on hippocampal neurogenesis in the context of brain aging.
Taurine treatment rescued cognitive deficits in APP/PS1 mice up to the age-matching wild-type mice in Y-maze and passive avoidance tests without modifying the behaviours of cognitively normal mice. In the cortex of APP/PS1 mice, taurine slightly decreased insoluble fraction of Aβ. While the exact mechanism of taurine in AD has not yet been ascertained, our results suggest that taurine can aid cognitive impairment and may inhibit Aβ-related damages.
In this study, we showed that chronic supplementation of taurine to aged mice significantly ameliorated the age-dependent decline in spatial memory acquisition and retention. These specific alterations of the inhibitory system caused by taurine treatment oppose those naturally occurring in the aging brain, suggesting a protective role of taurine in this process. An increased understanding of age-related neurochemical changes in the GABAergic system will be important in elucidating the underpinnings of the functional changes of aging. Taurine supplementation might help forestall the age-related decline in cognitive functions through interaction with the GABAergic system.
Improvement was observed in about 50% of the cases (21) both physically and psychologically. The physical improvement was noticed above all in the appearence of the skin while the psychological improvement was mostly related to both attention and memory.
Our results show that taurine increases sleep, while caffeine, as previously reported, attenuates sleep. Flies treated with both caffeine and taurine exhibit two differential effects which depend upon the ratio of taurine to caffeine. A high taurine:caffeine ratio promotes sleep, while a low ratio of taurine:caffeine inhibits sleep to a greater extent than the equivalent amount of caffeine alone.
Taurine also reversed the lower brain weight and improved the short-term memory in diabetic rats. Thus, the taurine antidepressant effect may be explained by interference with the GABA system, in line to its neuroprotective effect showed here by preventing brain weight loss and improving memory in diabetic rats.
In conclusion, our discoveries suggest that taurine supplementation has an antidepressant-like effect and an ability to change depression-related signaling cascades in the hippocampus.
Overall, these data suggest that TAU exerts an anxiolytic-like effect in zebrafish and the comparative analysis of behavior using different tasks is an interesting strategy for neuropsychiatric studies related to anxiety in this species.
- Muskelschwund
Increasing the taurine content of mdx muscle improved both in vivo and ex vivo muscle strength and function, potentially via anti-inflammatory and antioxidant effects of taurine.
Taurine appears to counteract atrophy by restoring regular microtubular apparatus and mitochondria and reducing the overload and the localization of autophagolysosomes
Tissue taurine depletion in taurine transporter knockout (TauTKO) mouse was found to shorten lifespan and accelerate skeletal muscle histological and functional defects
- Sexualhormonmangel
In addition, taurine and pioglitazone significantly decreased lipid peroxidation and DNA damage, and enhanced activity of the antioxidant enzymes in testes of diabetic rats. In conclusion, taurine and pioglitazone exerted protective effects against diabetes-induced testicular damage through attenuation of hyperglycemia, inflammation, oxidative stress and DNA damage, and up-regulation of the pituitary/gonadal axis.
Our data suggested that taurine can postpone testicular function deterioration in aged rats. Importantly, we observed obvious elevation of testicular antioxidant enzymes (SOD, GSH, GSH-Px) activities, and remarkable reduction of ROS and MDA by taurine administration, indicating taurine can decrease testicular oxidative stress and lipid peroxidation in aged rats. Finally, we found taurine effectively reduced testicular DNA fragmentation, increased testicular Bcl-2 protein expression, and decreased cytochrome c, Bax, Fas, FasL and caspase-3 expression, suggesting taurine can prohibit aged testicular apoptosis by mitochondrial dependent and independent signal pathway. In summary, our results indicated that taurine can suppress testicular function deterioration by increasing antioxidant ability and inhibiting apoptosis.
The results showed that taurine can significantly reduce the EL and ML; obviously increase the ERF, MF, IF, and EJF; stimulate the secretion of GnRH, LH, and T; and elevate penis NOS and NO level in aged rats. The results indicated that taurine can enhance the sexual response and mating ability in aged male rats by increasing the level of testosterone and NO
The present study demonstrated that a taurine supplement could stimulate the secretion of LH and T, increase the levels of testicular marker enzymes, elevate testicular antioxidation and improve sperm quality. The results imply that taurine plays important roles in male reproduction especially in aged animals.
- Entzündungen
In addition, taurine and pioglitazone significantly decreased lipid peroxidation and DNA damage, and enhanced activity of the antioxidant enzymes in testes of diabetic rats. In conclusion, taurine and pioglitazone exerted protective effects against diabetes-induced testicular damage through attenuation of hyperglycemia, inflammation, oxidative stress and DNA damage, and up-regulation of the pituitary/gonadal axis.
- Retinadegeneration
Taurine: the comeback of a neutraceutical in the prevention of retinal degenerations
We here review the evidence for a role of taurine in retinal ganglion cell survival and studies suggesting that this compound may be involved in the pathophysiology of glaucoma or diabetic retinopathy. Along with other antioxidant molecules, taurine should therefore be seriously reconsidered as a potential treatment for such retinal diseases.
- Haarausfall
Protective effects of taurine on human hair follicle grown in vitro.
We showed that taurine was taken up by the connective tissue sheath, proximal outer root sheath and hair bulb, promoted hair survival in vitro and prevented TGF-beta1-induced deleterious effects on hair follicle.
- Arthrosen
Chondroprotective effects of taurine in primary cultures of human articular chondrocytes
This study reveals that taurine is effective in proliferation promotion and phenotype maintenance of chondrocytes. Thus, taurine may be a useful pro-chondrogenic agent for autologous chondrocyte implantation in the treatment of cartilage repair.
- Fibrosen
When M cells were treated with various concentrations of taurine (10–50 mM), network formation and procollagen α1(I) expression were significantly suppressed in a dose dependent manner. Additionally, even in the presence of TGF-β1, taurine treatment effectively reduced the formation of a collagen fiber network. Additionally, even in the presence of TGF-β1, taurine treatment effectively reduced the formation of a collagen fiber network.
Reduced epidural fibrosis in vivo and decreased activation of fibroblasts in vitro after taurine treatment was mediated by EGR1. Taurine promises to be a potential prevention for epidural fibrosis after laminectomy.
Taurine supplementation significantly improved the liver pathologic findings, reduced the serum levels of aminotransferases and area of hepatic granuloma, and prevented fibrosis progression. In addition, taurine decreased the expression of the granulomatous and fibrogenic mediators transforming growth factor β1, tumor necrosis factor α, monocyte chemotactic protein 1α and macrophage inflammatory protein 1α as well as the endoplasmic reticulum stress marker glucose-regulated protein 78
Erectile function was significantly reduced in the diabetic rats compared with in the nondiabetic rats, and was improved in the diabetic rats treated with taurine. The corpus cavernosum of the rats with DED exhibited severe fibrosis and decreased smooth muscle content. Deposition of extracellular matrix proteins was increased in the diabetic rats, while expression of endothelial nitric oxide synthase/cyclic guanosine monophosphate/nitric oxide pathway-related proteins was reduced. Taurine supplementation ameliorated erectile response as well as histologic and molecular alterations.
- Verletzungen
The numerical density of the fibroblasts, volume density of the collagen bundles, and length densities of the vessels in groups T1 and T2 were significantly higher than in group C (P < 0.05). The fibroblast numerical density of group T1 was higher than that of group T2 (P = 0.02). Tu showed the ability to improve the wound healing process and tissue regeneration
15.1 Sehnenverletzungen
Effect of taurine on rat Achilles tendon healing
After histological assessment, we found that fibroblast proliferation, edema, and inflammation statistically decreased in the treatment group (p < 0.05). These findings could indicate greater tendon strength with less adhesion formation, and taurine may have an effect on adhesion formation.
- Alterung
Tissue taurine depletion in taurine transporter knockout (TauTKO) mouse was found to shorten lifespan and accelerate skeletal muscle histological and functional defects, including an increase in central nuclei containing myotubes, a reduction in mitochondrial complex 1 activity and an induction in an aging biomarker, Cyclin-dependent kinase 4 inhibitor A (p16INK4a).
Evidence accumulating from the studies with taurine transporter knockout (TauTKO) mouse has indicated that tissue taurine depletion led to aging-associated disorders in several tissues, including heart, skeletal muscle, liver, skin, and shortened the lifespan. Moreover, muscle taurine depletion causes ER stress to activate unfolded protein response. In conclusion, endogenous taurine acts as an anti-aging molecule via, in part, proper protein folding property.
The results indicated that the MLS of drosophila in high dose group and the MMLS of female in low, middle and high doses increased compared with control group. The delaying senility function of taurine was preliminarily evidenced by this research.
In this study, we showed that chronic supplementation of taurine to aged mice significantly ameliorated the age-dependent decline in spatial memory acquisition and retention. These specific alterations of the inhibitory system caused by taurine treatment oppose those naturally occurring in the aging brain, suggesting a protective role of taurine in this process. An increased understanding of age-related neurochemical changes in the GABAergic system will be important in elucidating the underpinnings of the functional changes of aging. Taurine supplementation might help forestall the age-related decline in cognitive functions through interaction with the GABAergic system.
- Krebs
The administration of taurine significantly reduced the DMBA-induced breast cancer rate from 80% to 40% in rats (p<0.05). We propose that these differential metabolites may be potential biomarkers for monitoring cancer therapy and prognosis in the clinic. This study provides a scientific basis for further investigations of the antitumor mechanism of taurine and the development of novel therapeutic strategies to treat breast cancer.
17.1 Brustkrebs
Taurine (100 mg/kg body weight) treatment decreased liver mitochondrial LPO and augmented the activities/levels of enzymic, and non-enzymic antioxidants, tricarboxylic acid cycle enzymes and ETC complexes. The results of our present study demonstrated the chemotherapeutic efficacy of taurine treatment for DMBA-induced breast carcinomas.
Further studies showed that Tau promoted apoptosis in human breast cancer cells and inhibited the growth of tumor in nude mice by inducing the expression of PUMA, which further up- and downregulated the expression of Bax and Bcl-2 protein, giving rise to increased activation of caspase-3. Collectively, these results indicate that Tau is a potent candidate for the chemotherapy of breast cancer through increasing the PUMA expression independent of p53 status.
17.2. Darmkrebs
Mechanism of taurine-induced apoptosis in human colon cancer cells
As a result, apoptosis was decreased in response to Tau treatment. All these results indicated that PUMA plays a critical role in Tau-induced apoptosis pathway in human colorectal cancer cells. Demonstration of the molecular mechanism involved in the anti-tumor effect of Tau may be useful in the therapeutic target selection for p53-deficient colorectal cancer.
17.3. Leukämie
This work highlights the possibility of using curcumin and taurine as a potential useful therapy in the management of patients suffering from chronic and acute myeloid leukemias.
- Darmdysbiose
Effects of taurine on gut microbiota and metabolism in mice
The results showed that the effect of taurine on gut microbiota could reduce the abundance of Proteobacteria, especially Helicobacter. Moreover, we found that the SCFA content was increased in feces of the NE group while LPS content was decreased in serum of the NE group; the SOD activity in serum and livers of the NE and CS groups were not changed significantly compare to that of the CK group. In conclusion, taurine could regulate the gut micro-ecology, which might be of benefit to health by inhibiting the growth of harmful bacteria, accelerating the production of SCFA and reducing LPS concentration.
- Toxische Hepatitis
The inducible nitric oxide synthase (iNOS), C-reactive protein (CRP), tumor necrosis factors (TNF)-α, interleukin (IL)-6, and IL-1β were also significantly lower in the Tau and Sil groups than in the Alc group. The experimental results indicated that hepatoprotection against alcohol-induced inflammation may be mediated by decreased TLR-4/MyD88 signaling.
- Perioodontitis
Based on the biochemical and clinical assessments, taurine seems to exert a protective role against the oxidative stress in the management of patients with chronic periodontitis.
- Nebenwirkungen Chemotherapie
Taurine attenuates chemotherapy-induced nausea and vomiting in acute lymphoblastic leukemia
The results indicated that taurine-supplemented patients reported a significant (P < 0.05) improvement in chemotherapy-induced nausea and/or vomiting after taking taurine during study. Taurine significantly improved chemotherapy-induced taste and smell alterations (P < 0.05). Moreover, taurine significantly reduced weariness compared to placebo group (P < 0.05). This study showed that taurine co-administration decreased chemotherapy-induced nausea and vomiting during the maintenance therapy in acute lymphoblastic leukemia.
- Osteoporose
Vitamin B₁₂-dependent taurine synthesis regulates growth and bone mass
Taurine increased GH-dependent IGF1 synthesis in the liver, which subsequently enhanced osteoblast function, and in B12-deficient offspring, oral administration of taurine rescued their growth retardation and osteoporosis phenotypes. These results identify B12 as an essential vitamin that positively regulates postweaning growth and bone formation through taurine synthesis and suggests potential therapies to increase bone mass
- Pankreatitis
The protective effects of taurine on experimental acute pancreatitis in a rat model
These results showed that Tau reduces lipid peroxidation, amylase and MPO activities and the concentrations of proinflammatory cytokines secondary to AP and also increases superoxide dismutase and glutathione peroxidase activities in rats with sodium taurocholate-induced AP. It also has a marked ameliorative effect at histopathologic lesions. With these effects, Tau protects the cells from oxidative damage, reduces inflammation and promotes regression of pancreatic damage.
- Thrombosen
Our findings suggest that taurine enhances endogenous thrombolytic activity which could be a mechanism of the earlier observed cardioprotective and antithrombotic effect.
Wirkungen:
Taurine bewirkt Durchblutungsförderung durch Minderung osmotischer Barrieren.
Es reduziert Narbenbildung und Fibrosen.
Es wirkt antientzündlich und antioxidativ.
Es wirkt Organprotektiv
Taurine plays an important role in brain development, including neuronal proliferation, stem cell proliferation, and differentiation, via several mechanisms. Taurine can be directly used in clinical applications to improve brain development because it has no toxic effects on humans.
A reduction in taurine content accompanies the ageing process in many tissues. In fact, the decline of brain taurine levels has been associated with cognitive deficits whereas chronic administration of taurine seems to ameliorate age-related deficits such as memory acquisition and retention.
Taurine can act as an antioxidant, a testosterone secretion stimulator, a sperm membrane stabilizer and motility factor, and an anti-apoptotic agent.
In all animals taurine provoked the disappearance of EEG epileptic abnormalities.
Symptoms of taurine deficiency include: anxiety, epilepsy, hyperactivity, and impaired brain function. Low levels of the amino acid cysteine and vitamin B6 can cause taurine deficiency.
Taurine protects cell membranes from damage, and enhances the immune system by stimulating the release of interleukin-1 in macrophages, and increasing the phagocytic and bactericidal activity of neutrophils. It also helps to detoxify toxic substances such as retinoids and environmental toxins. Several studies have found that taurine might be useful in the treatment of congestive heart failure (CHF), and at least one study has found that it may also be useful for acute viral hepatitis. Taurine also increases levels of the neurotransmitter acetylcholine levels and helps to regulate the nervous system.
Taurin wird klinisch zur Behandlung von Herz-Kreislauf-Erkrankungen, Hypercholesterinämie, Anfallsleiden, Augenerkrankungen, Diabetes mellitus, Alzheimer-Krankheit, Leberfunktionsstörungen, Mukoviszidose und Alkoholismus eingesetzt.
One of its main functions is osmoregulation; however, it also exhibits cytoprotective activity by diminishing injury caused by stress and disease.
Congestive Heart Failure ; Viral Hepatitis
Alcoholism ; Cataracts ; Diabetes ; Epilepsy ; Gallbladder Disease ; Hypertension (High Blood Pressure) ; Multiple Sclerosis ; Psoriasis ; Stroke
Taurin stimuliert GABA, beruhigt die Nerven und baut Stresshormone ab.
Taurine is described as being a cellular activator for regulating hair cells and is proposed in hair-stimulating compositions for topical application.
Taurine plays a vital role in hearing. In fact, studies have found that in some cases, taurine can reverse the biochemical processes behind hearing loss.53,54 Other studies have demonstrated that taurine can almost completely eliminate the ringing in the ears associated with tinnitus.
Artikel
What about taurine
http://afibbers.org/resources/taurine.pdf
The Benefits of Taurine
http://main.poliquingroup.com/articlesmultimedia/articles/article/782/ten_benefits_of_taurine.aspx
Taurine: Important Brain Nutrient
http://www.life-enhancement.com/magazine/article/2879-taurine-important-brain-nutrient
The Forgotten Longevity Benefits of Taurine
http://www.lifeextension.com/magazine/2013/6/the-forgotten-longevity-benefits-of-taurine/page-01
The Multiple Benefits of Taurine
https://www.victoriahealth.com/editorial/the-multiple-benefits-of-taurine
TCM
Die 5-Elementeanalyse bildet die Energiedynamik von Taurin wie folgt ab:
Schwarz : Studien
Rot: Studien Hauptwirkung
Zum Verständnis der Abkürzungen des Schaubildes siehe
Struktur – und Wandlungsmodell der chinesischen Medizin
http://www.chinesischemedizin.com/CMS/index.php/struktur-und-wandlungsmodell.html
Chinesische Medizin 
dank je wel
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Verfasst von Henry Ernawan | 6. März 2018, 5:07